时间标签：数据日期 2026-11-05 · 生成时间 2026-11-05 05:30（Asia/Shanghai）｜ 检索窗口 PubMed edat 2026/11/02 – 2026/11/06 ｜ 情报发生日期 2026-11-05
证据级别：摘要级。本条依据 PubMed 结构化摘要与元数据撰写，付费墙全文未获取、未逐节核验，本站未做任何数值推算补全。
摘要（PubMed 原文节选）
The precursor of Methcathinone (MeCa) is a cathinone obtained from the plant Catha edulis Forsk. As one of the most prevalent novel psychoactive substances, MeCa has severe neurotoxicology and contributes cognitive dysfunction. Currently, no effective method is available for stopping its progress. Ferroptosis has been proven to contribute…
对本项目的价值
参考价值：属「营养感知—自噬—蛋白稳态轴」方向；mTOR / 自噬 / 蛋白质稳态栏目可用作本项目文献库与综述素材。 ｜ 价值分 1.8/5（★★☆☆☆）
全文不可及原因
无 PMCID 且 Europe PMC / OpenAlex / Unpaywall 未提供可下载 OA 全文
原文题录
- 中文题名：吡哆醇经 Nrf2/GPX4 通路抑制铁死亡减轻甲基卡西酮诱导认知障碍
- 英文标题：Pyridoxine attenuates Methcathinone-induced cognitive impairment by suppressing ferroptosis via Nrf2/GPX4 pathway.
- 期刊与标识：Experimental neurology · PMID 42977018 · DOI 10.1016/j.expneurol.2026.116093