时间标签：数据日期 2026-11-05 · 生成时间 2026-11-05 05:30（Asia/Shanghai）｜ 检索窗口 PubMed edat 2026/11/02 – 2026/11/06 ｜ 情报发生日期 2026-11-05
证据级别：摘要级。本条依据 PubMed 结构化摘要与元数据撰写，付费墙全文未获取、未逐节核验，本站未做任何数值推算补全。
摘要（PubMed 原文节选）
BACKGROUND: Rotator cuff repair failures are primarily driven by intrinsic tissue degeneration. While mitochondrial dysfunction is implicated in this cascade, the specific metabolic signals driving structural tendon failure remain poorly defined. The succinate-SUCNR1 axis may function as a critical metabolic checkpoint in this process. HY…
对本项目的价值
参考价值：属「衰老细胞清除 / SASP 轴」方向；细胞衰老 / senolytics / SASP栏目可用作本项目文献库与综述素材。 ｜ 价值分 1.8/5（★★☆☆☆）
全文不可及原因
无 PMCID 且 Europe PMC / OpenAlex / Unpaywall 未提供可下载 OA 全文
原文题录
- 中文题名：阻断琥珀酸-SUCNR1 代谢轴有助于恢复线粒体稳态并预防小鼠过度使用性肩袖肌腱病结构失效
- 英文标题：Blockade of the Succinate-SUCNR1 Metabolic Axis Helps Restore Mitochondrial Homeostasis and Prevents Structural Failure in Overuse-Induced Rotator Cuff Tendinopathy in a Mouse Model.
- 期刊与标识：The American journal of sports medicine · PMID 42975033 · DOI 10.1177/03635465261494256