时间标签:数据日期 2026-11-05 · 生成时间 2026-11-05 05:30(Asia/Shanghai)| 检索窗口 PubMed edat 2026/11/02 – 2026/11/06 | 情报发生日期 2026-11-05
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摘要(PubMed 原文节选)
BACKGROUND: Imbalanced bone formation and resorption is the central pathological feature of osteoporosis, while chronic low-grade inflammation contributes to progressive disruption of the bone marrow microenvironment. Emerging evidence suggests that NLRP3 inflammasome-mediated pyroptosis may amplify local inflammation by promoting the rel…
对本项目的价值
参考价值:属「营养感知—自噬—蛋白稳态轴」方向;mTOR / 自噬 / 蛋白质稳态栏目可用作本项目文献库与综述素材。 | 价值分 1.8/5(★★☆☆☆)
全文不可及原因
无 PMCID 且 Europe PMC / OpenAlex / Unpaywall 未提供可下载 OA 全文
原文题录
- 中文题名:骨质疏松中的自噬-细胞焦亡串扰:将线粒体稳态与炎性骨重塑相连
- 英文标题:Autophagy-pyroptosis crosstalk in osteoporosis: Linking mitochondrial homeostasis to inflammatory bone remodeling.
- 期刊与标识:Tissue & cell · PMID 42971078 · DOI 10.1016/j.tice.2026.104101