时间标签:数据日期 2026-11-05 · 生成时间 2026-11-05 05:30(Asia/Shanghai)| 检索窗口 PubMed edat 2026/11/02 – 2026/11/06 | 情报发生日期 2026-11-05
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摘要(PubMed 原文节选)
BACKGROUND: Glioblastoma (GBM) exhibits profound therapeutic resistance that is largely attributed to its remarkable cellular plasticity and capacity to adapt to therapeutic and environmental stress. Although suppression of mechanistic target of rapamycin (mTOR) signaling has been extensively investigated as a therapeutic strategy, the ad…
对本项目的价值
参考价值:属「营养感知—自噬—蛋白稳态轴」方向;mTOR / 自噬 / 蛋白质稳态栏目可用作本项目文献库与综述素材。 | 价值分 1.8/5(★★☆☆☆)
全文不可及原因
无 PMCID 且 Europe PMC / OpenAlex / Unpaywall 未提供可下载 OA 全文
原文题录
- 中文题名:适应性诱导 NFS1 将 mTOR 抑制与胶质母细胞瘤干性相关可塑性相连
- 英文标题:Adaptive induction of NFS1 links mTOR suppression to stemness-associated plasticity in glioblastoma.
- 期刊与标识:Journal of neuro-oncology · PMID 42975782 · DOI 10.1007/s11060-026-05856-1